Publikationsansicht

Toll-like receptors (TLR) 4 and 2 regulate the innate immune response. Study of endotoxin influence in mice (2004)

Abstract
TOLL LIKE RECEPTORS TLR AND REGULATE THE INNATE IMMUNE RESPONSE Study endotoxin influence mice KIR HAR Department Paediatrics Biocenter Oulu University Oulu OULU Abstract Finnish KIRSI HARJU TOLL LIKE RECEPTORS TLR AND REGULATE THE INNATE IMMUNE RESPONSE Study endotoxin influence mice Academic Dissertation presented with the assent the Faculty Medicine University Oulu for public discussion the Auditorium Oulu University Hospital May noon L L I U Copyright University Oulu Supervised Professor Mikko Hallman Doctor Marja Ojaniemi Doctor Virpi Glumoff Reviewed Professor Outi Vaarala Professor Olli Vainio ISBN nid ISBN PDF http herkules oulu isbn ISSN http herkules oulu issn OULU UNIVERSITY PRESS OULU Harju Kirsi Toll like receptors TLR and regulate the innate immune response Study endotoxin influence mice Department Paediatrics Biocenter Oulu University Oulu Box FIN University Oulu Finland Oulu Finland Abstract The response the innate immune system triggered through Toll like transmembrane receptors TLR that recognize variety microbial products TLR the principal mediator for Gram negative bacterial endotoxin LPS whereas TLR mediates the response Gram positive bacteria mycobacteria and yeast Stimulation TLR activates complex cascades leading first the production inflammatory mediators such proinflammatory cytokines and TNF Overproduction inflammatory cytokines well failure the activation innate immunity are detrimental the host Excess inflammatory stimulation leads septic shock wh

Details der Publikation
Download http://herkules.oulu.fi/isbn9514273257/
Herausgeber University of Oulu
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Keywords infant; premature, inflammation, lipopolysaccharides, receptors; cell surface, signal transduction, ennenaikainen syntymä, lipopolysakkaridi, solukalvoreseptorit, solusignalointi, synnynnäinen immuniteetti, cytokines, immunity; natural, sytokiinit, tulehdus
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Sprache eng

Literaturangaben in der Publikation (7)
Antenatal infection/inflammation and postnatal lung maturation and injury (2001)
Genetic control of the innate immune response (2003)
Phylogenetic variation and polymorphism at the Toll-like receptor 4 locus (TLR4) (2000)
Toll-Like Receptor 4 (TLR4)-Deficient Murine Macrophage Cell Line as an In Vitro Assay System To Show TLR4-Independent Signaling of Bacteroides fragilis Lipopolysaccharide
Binding of lipopolysaccharide (LPS) to an 80-kilodalton membrane protein of human cells is mediated by soluble CD14 and LPS-binding protein.
Interleukin-18 (IFNgamma-inducing factor) induces IL-8 and IL-1beta via TNFalpha production from non-CD14+ human blood mononuclear cells.
Differential roles of Toll-like receptors in the elicitation of proinflammatory responses by macrophages