James G. Conway

Details der Publikationsliste

Zeitraum

1987 - 1989

Anzahl

4

Co-Autoren

Relationship of oxidative damage to the hepatocarcinogenicity of the peroxisome proliferators di(2-ethylhexyl)phthalate and Wy-14,643 (1989)

Conway, James G., Tomaszewski, Konrad E., Olson, Michael J., Cattley, Russell C., Marsman, Daniel S., Popp, James A.

Quantitative comparisons of the time course of biochemical and morphological changes induced by peroxisome proliferators resulting in low and high incidences of hepatic cancer have not been conducted...

Carcinogen treatment increases glutathione hydrolysis by {gamma}-glutamyl transpeptidase (1987)

Conway, James G., Neptun, Douglas A., Garvey, Linda K., Popp, James A.

The effect of carcinogen treatment on γ-glutamyl transpep-tidase (GGT)-mediated hydrolysis of GSH to glutamate and cysteinylglycine in the blood and bile compartments was investigated in livers...

Inhibition of colony-stimulating-factor-1 signaling in vivo with the orally bioavailable cFMS kinase inhibitor GW2580

Conway, James G., McDonald, Brad, Parham, Janet, Keith, Barry, Rusnak, David W., Shaw, Eva, ...

Colony-stimulating-factor-1 (CSF-1) signaling through cFMS receptor kinase is increased in several diseases. To help investigate the role of cFMS kinase in disease, we identified GW2580, an orally...

Inhibition of colony-stimulating-factor-1 signaling in vivo with the orally bioavailable cFMS kinase inhibitor GW2580

Conway, James G., McDonald, Brad, Parham, Janet, Keith, Barry, Rusnak, David W., Shaw, Eva, ...

Colony-stimulating-factor-1 (CSF-1) signaling through cFMS receptor kinase is increased in several diseases. To help investigate the role of cFMS kinase in disease, we identified GW2580, an orally...