Karl Münger

Details der Publikationsliste

Zeitraum

1987 - 2005

Anzahl

44

Co-Autoren

Excessive centrosome abnormalities without ongoing numerical chromosome instability in a Burkitt's lymphoma (2003)

Duensing, Stefan, Lee, Benjamin H, Cin, Paola, Münger, Karl

Abstract Numerical and structural centrosome abnormalities are detected in various human malignancies and have been implicated in the formation of multipolar mitoses, chromosome missegregation, and...

Expression of the synthetic gene of an artificial DDT-binding polypeptide in Escherichia coli (1987)

Moser, Rudolf, Frey, Stefan, Münger, Karl, Hehlgans, Thomas, Klauser, Stephan, Langen, Hanno, ...

This paper reports the expression of an artificial functional polypeptide in bacteria. The gene of a designed 24-residue DDT-binding polypeptide (DBP) was inserted between the BamHI and PstI cleavage...

TID1, a human homolog of the Drosophila tumor suppressor l(2)tid, encodes two mitochondrial modulators of apoptosis with opposing functions

Syken, Josh, De-Medina, Tali, Münger, Karl

Mitochondria have emerged as central regulators of apoptosis. Here, we show that TID1, a human homolog of the Drosophila tumor suppressor lethal (2) tumorous imaginal discs, l(2)tid, encodes two...

Immortalization of primary human keratinocytes by the helix–loop–helix protein, Id-1

Alani, Rhoda M., Hasskarl, Jens, Grace, Miranda, Hernandez, Maria-Clementia, Israel, Mark A., Münger, Karl

Basic helix–loop–helix (bHLH) DNA-binding proteins have been demonstrated to regulate tissue-specific transcription within multiple cell lineages. The Id family of helix–loop–helix proteins...

The human papillomavirus type 16 E6 and E7 oncoproteins cooperate to induce mitotic defects and genomic instability by uncoupling centrosome duplication from the cell division cycle

Duensing, Stefan, Lee, Lily Y., Duensing, Anette, Basile, John, Piboonniyom, Siribang-on, Gonzalez, Sonia, ...

Loss of genomic integrity is a defining feature of many human malignancies, including human papillomavirus (HPV)-associated preinvasive and invasive genital squamous lesions. Here we show that...

Degradation of the Retinoblastoma Tumor Suppressor by the Human Papillomavirus Type 16 E7 Oncoprotein Is Important for Functional Inactivation and Is Separable from Proteasomal Degradation of E7

Gonzalez, Sonia L., Stremlau, Matt, He, Xi, Basile, John R., Münger, Karl

The steady-state level and metabolic half-life of retinoblastoma tumor suppressor protein pRB are decreased in cells that express high-risk human papillomavirus (HPV) E7 proteins. Here we show that...

Centrosome Abnormalities and Genomic Instability by Episomal Expression of Human Papillomavirus Type 16 in Raft Cultures of Human Keratinocytes

Duensing, Stefan, Duensing, Anette, Flores, Elsa R., Do, Anh, Lambert, Paul F., Münger, Karl

Primary human keratinocytes with ectopic expression of high-risk human papillomavirus (HPV) E6 and E7 oncoproteins display abnormal centrosome numbers, multipolar mitoses, and aneusomy. However, it...

Quantitative Role of the Human Papillomavirus Type 16 E5 Gene during the Productive Stage of the Viral Life Cycle

Genther, Sybil M., Sterling, Stephanie, Duensing, Stefan, Münger, Karl, Sattler, Carol, Lambert, Paul F.

Human papillomaviruses (HPVs) are small circular DNA viruses that cause warts. Infection with high-risk anogenital HPVs, such as HPV type 16 (HPV16), is associated with human cancers, specifically...

Excessive centrosome abnormalities without ongoing numerical chromosome instability in a Burkitt's lymphoma

Duensing, Stefan, Lee, Benjamin H, Cin, Paola Dal, Münger, Karl

Numerical and structural centrosome abnormalities are detected in various human malignancies and have been implicated in the formation of multipolar mitoses, chromosome missegregation, and...

Human Papillomavirus Type 16 E7 Oncoprotein Can Induce Abnormal Centrosome Duplication through a Mechanism Independent of Inactivation of Retinoblastoma Protein Family Members

Duensing, Stefan, Münger, Karl

The human papillomavirus type 16 (HPV-16) E7 oncoprotein rapidly induces centrosome duplication errors in primary human cells, thereby increasing the propensity for multipolar mitoses, which can lead...

Recapitulation of the Effects of the Human Papillomavirus Type 16 E7 Oncogene on Mouse Epithelium by Somatic Rb Deletion and Detection of pRb-Independent Effects of E7 In Vivo

Balsitis, Scott J., Sage, Julien, Duensing, Stefan, Münger, Karl, Jacks, Tyler, Lambert, Paul F.

Although the human papillomavirus (HPV) E7 oncogene is known to contribute to the development of human cervical cancer, the mechanisms of its carcinogenesis are poorly understood. The first...

The human papillomavirus E7 oncoprotein can uncouple cellular differentiation and proliferation in human keratinocytes by abrogating p21Cip1-mediated inhibition of cdk2

Jones, D. Leanne, Alani, Rhoda M., Münger, Karl

The high risk human papillomaviruses (HPVs) are associated etiologically with the majority of human cervical carcinomas. These HPVs encode two viral oncoproteins, E6 and E7, which are expressed...

Association of the human papillomavirus type 16 E7 oncoprotein with the 600-kDa retinoblastoma protein-associated factor, p600

Huh, Kyung-Won, DeMasi, Joseph, Ogawa, Hidesato, Nakatani, Yoshihiro, Howley, Peter M., Münger, Karl

The human papillomavirus type 16 (HPV-16) E7 gene encodes a multifunctional oncoprotein that can subvert multiple cellular regulatory pathways. The best-known cellular targets of the HPV-16 E7...

Bovine papillomavirus E7 transformation function correlates with cellular p600 protein binding

DeMasi, Joseph, Huh, Kyung-Won, Nakatani, Yoshihiro, Münger, Karl, Howley, Peter M.

The E7 oncoprotein of bovine papillomavirus type 1 (BPV-1) is required for the full transformation activity of the virus. However, the mechanism by which E7 contributes to cellular transformation is...

The Human DEK Proto-Oncogene Is a Senescence Inhibitor and an Upregulated Target of High-Risk Human Papillomavirus E7

Wise-Draper, Trisha M., Allen, Hillary V., Thobe, Megan N., Jones, Elizabeth E., Habash, Kristen B., Münger, Karl, ...

The human DEK proto-oncogene is a nucleic acid binding protein with suspected roles in human carcinogenesis, autoimmune disease, and viral infection. Intracellular DEK functions, however, are poorly...

Human Papillomavirus E7 Oncoprotein Dysregulates Steroid Receptor Coactivator 1 Localization and Function

Baldwin, Amy, Huh, Kyung-Won, Münger, Karl

High-risk human papillomaviruses (HPVs) are present in virtually all cervical carcinomas. However, the majority of women infected with high-risk HPVs do not develop cervical cancer. Therefore,...

TID1, a human homolog of the Drosophila tumor suppressor l(2)tid, encodes two mitochondrial modulators of apoptosis with opposing functions

Syken, Josh, De-Medina, Tali, Münger, Karl

Mitochondria have emerged as central regulators of apoptosis. Here, we show that TID1, a human homolog of the Drosophila tumor suppressor lethal (2) tumorous imaginal discs, l(2)tid, encodes two...

Immortalization of primary human keratinocytes by the helix–loop–helix protein, Id-1

Alani, Rhoda M., Hasskarl, Jens, Grace, Miranda, Hernandez, Maria-Clementia, Israel, Mark A., Münger, Karl

Basic helix–loop–helix (bHLH) DNA-binding proteins have been demonstrated to regulate tissue-specific transcription within multiple cell lineages. The Id family of helix–loop–helix proteins...

The human papillomavirus type 16 E6 and E7 oncoproteins cooperate to induce mitotic defects and genomic instability by uncoupling centrosome duplication from the cell division cycle

Duensing, Stefan, Lee, Lily Y., Duensing, Anette, Basile, John, Piboonniyom, Siribang-on, Gonzalez, Sonia, ...

Loss of genomic integrity is a defining feature of many human malignancies, including human papillomavirus (HPV)-associated preinvasive and invasive genital squamous lesions. Here we show that...

Degradation of the Retinoblastoma Tumor Suppressor by the Human Papillomavirus Type 16 E7 Oncoprotein Is Important for Functional Inactivation and Is Separable from Proteasomal Degradation of E7

Gonzalez, Sonia L., Stremlau, Matt, He, Xi, Basile, John R., Münger, Karl

The steady-state level and metabolic half-life of retinoblastoma tumor suppressor protein pRB are decreased in cells that express high-risk human papillomavirus (HPV) E7 proteins. Here we show that...

Centrosome Abnormalities and Genomic Instability by Episomal Expression of Human Papillomavirus Type 16 in Raft Cultures of Human Keratinocytes

Duensing, Stefan, Duensing, Anette, Flores, Elsa R., Do, Anh, Lambert, Paul F., Münger, Karl

Primary human keratinocytes with ectopic expression of high-risk human papillomavirus (HPV) E6 and E7 oncoproteins display abnormal centrosome numbers, multipolar mitoses, and aneusomy. However, it...

Quantitative Role of the Human Papillomavirus Type 16 E5 Gene during the Productive Stage of the Viral Life Cycle

Genther, Sybil M., Sterling, Stephanie, Duensing, Stefan, Münger, Karl, Sattler, Carol, Lambert, Paul F.

Human papillomaviruses (HPVs) are small circular DNA viruses that cause warts. Infection with high-risk anogenital HPVs, such as HPV type 16 (HPV16), is associated with human cancers, specifically...

Excessive centrosome abnormalities without ongoing numerical chromosome instability in a Burkitt's lymphoma

Duensing, Stefan, Lee, Benjamin H, Cin, Paola Dal, Münger, Karl

Numerical and structural centrosome abnormalities are detected in various human malignancies and have been implicated in the formation of multipolar mitoses, chromosome missegregation, and...

Human Papillomavirus Type 16 E7 Oncoprotein Can Induce Abnormal Centrosome Duplication through a Mechanism Independent of Inactivation of Retinoblastoma Protein Family Members

Duensing, Stefan, Münger, Karl

The human papillomavirus type 16 (HPV-16) E7 oncoprotein rapidly induces centrosome duplication errors in primary human cells, thereby increasing the propensity for multipolar mitoses, which can lead...

Recapitulation of the Effects of the Human Papillomavirus Type 16 E7 Oncogene on Mouse Epithelium by Somatic Rb Deletion and Detection of pRb-Independent Effects of E7 In Vivo

Balsitis, Scott J., Sage, Julien, Duensing, Stefan, Münger, Karl, Jacks, Tyler, Lambert, Paul F.

Although the human papillomavirus (HPV) E7 oncogene is known to contribute to the development of human cervical cancer, the mechanisms of its carcinogenesis are poorly understood. The first...

The human papillomavirus E7 oncoprotein can uncouple cellular differentiation and proliferation in human keratinocytes by abrogating p21Cip1-mediated inhibition of cdk2

Jones, D. Leanne, Alani, Rhoda M., Münger, Karl

The high risk human papillomaviruses (HPVs) are associated etiologically with the majority of human cervical carcinomas. These HPVs encode two viral oncoproteins, E6 and E7, which are expressed...

Association of the human papillomavirus type 16 E7 oncoprotein with the 600-kDa retinoblastoma protein-associated factor, p600

Huh, Kyung-Won, DeMasi, Joseph, Ogawa, Hidesato, Nakatani, Yoshihiro, Howley, Peter M., Münger, Karl

The human papillomavirus type 16 (HPV-16) E7 gene encodes a multifunctional oncoprotein that can subvert multiple cellular regulatory pathways. The best-known cellular targets of the HPV-16 E7...

Bovine papillomavirus E7 transformation function correlates with cellular p600 protein binding

DeMasi, Joseph, Huh, Kyung-Won, Nakatani, Yoshihiro, Münger, Karl, Howley, Peter M.

The E7 oncoprotein of bovine papillomavirus type 1 (BPV-1) is required for the full transformation activity of the virus. However, the mechanism by which E7 contributes to cellular transformation is...

The Human DEK Proto-Oncogene Is a Senescence Inhibitor and an Upregulated Target of High-Risk Human Papillomavirus E7

Wise-Draper, Trisha M., Allen, Hillary V., Thobe, Megan N., Jones, Elizabeth E., Habash, Kristen B., Münger, Karl, ...

The human DEK proto-oncogene is a nucleic acid binding protein with suspected roles in human carcinogenesis, autoimmune disease, and viral infection. Intracellular DEK functions, however, are poorly...

Human Papillomavirus E7 Oncoprotein Dysregulates Steroid Receptor Coactivator 1 Localization and Function

Baldwin, Amy, Huh, Kyung-Won, Münger, Karl

High-risk human papillomaviruses (HPVs) are present in virtually all cervical carcinomas. However, the majority of women infected with high-risk HPVs do not develop cervical cancer. Therefore,...

Human Papillomavirus Type 16 E7 Oncoprotein Associates with the Centrosomal Component γ-Tubulin▿

Nguyen, Christine L., Eichwald, Catherine, Nibert, Max L., Münger, Karl

Expression of a high-risk human papillomavirus (HPV) E7 oncoprotein is sufficient to induce aberrant centrosome duplication in primary human cells. The resulting centrosome-associated mitotic...

Human Papillomavirus Type 16 E7 Oncoprotein Associates with E2F6▿

McLaughlin-Drubin, Margaret E., Huh, Kyung-Won, Münger, Karl

The papillomavirus life cycle is intimately coupled to the differentiation state of the infected epithelium. Since papillomaviruses lack most of the rate-limiting enzymes required for genome...

Kinase requirements in human cells: II. Genetic interaction screens identify kinase requirements following HPV16 E7 expression in cancer cells

Baldwin, Amy, Li, Wenliang, Grace, Miranda, Pearlberg, Joseph, Harlow, Ed, Münger, Karl, ...

Human papillomavirus (HPV) oncoproteins subvert cellular signaling pathways, including kinase pathways, during the carcinogenic process. To identify kinases targeted by the HPV16 E7 oncoprotein,...

Human Papillomavirus E7 Protein Deregulates Mitosis via an Association with Nuclear Mitotic Apparatus Protein 1 ▿

Nguyen, Christine L., Münger, Karl

We previously observed that high-risk human papillomavirus type 16 (HPV16) E7 expression leads to the delocalization of dynein from mitotic spindles (C. L. Nguyen, M. E. McLaughlin-Drubin, and K....